Wednesday, July 6, 2022

Biden DOJ Sues To Block Arizona's Proof Of Citizenship Voting Law

AVOID COMMERCIAL SEED OILS

The above clip is taken from the documentary, Fat Fiction.  The clip above covers the section between 1:18:30 to 1:20:45.

 

1:20:50  

Nina TiecholzThe Minnesota Coronary Experiment, MCE, 1968-1973, was the biggest test of Ancel Keyes' hypothesis.  It took place at 6 Minnesota mental hospitals and 1 nursing home, which is a kind of experiment you can't do anymore because it's considered unethical, but back then it had the benefit of being highly controlled.  This means you're feeding people all their food, so you know what they're eating, and they can't get outside food, so they can't cheat. 

Andrew Mente:  In the diet, they replaced saturated fat with polyunsaturated fat (PUSFs), an intervention.  What they found was that with the intervention, cholesterol levels went down, which is what you'd expect, but mortality actually trended up.  

Gary Taubes:  Turned out, that the people who were on the cholesterol-lowering diet had more heart disease, and more deaths than the people eating the controlled diet, which was the exact opposite of 

Andrew Mente:  So, it did not support the [Keys'] diet-heart hypothesis. 

Gary Taubes:  But they never published the results.  

Not publishing the results, is scientific fraud.  

The Heart Association didn't come out and say, "Sorry, we were wrong."  

from the Show Notes: 

Dr. Mark Hyman, Functional Medicine Doctor and Director of the Cleveland Clinic Center for Functional Medicine and the UltraWellness Center and Chairman of the board of the Institute for Functional Medicine. 

Dr. Sarah Hallberg, Obesity Expert has reversed Type 2 Diabetes in hundreds of patients by ignoring the guidelines and prescribing a high fat, low carb nutrition plan. 

Dr. Jason Fung, Nephrologist and author of The Obesity Code: Unlocking the Secrets of Weight Loss (Why Intermittent Fasting Is the Key to Controlling Your Weight), a book for reversing Type 2 Diabetes with LCHF and Intermittent Fasting. 

Professor Tim Noakes, the South African author of the Lore of Running, called low-fat diets "genocide." 

Nina Teicholz, Journalist and author of the Big Fat Surprise.  On her Twitter feed, she features the book, Ravenous: Otto Warburg, the Nazis, and the Search for the Cancer-Diet Connection, Sam Apple, 2021. 

Gary Taubes, Journalist and author of Good Calories, Bad Calories and The Case Against Sugar.  

Dr. Rob Lustig, Pediatric Endocrinologist at University of California, San Francisco. 

Dr. Bret Scher, Cardiologist and Lipidologist practicing in San Diego 

Dr. Eric Westman, Director of the Lifestyle Medicine Clinic at Duke University Medical Center in Durham, North Carolina.  

Dr. Brian Lenzkes, Internal Medicine. 

Doctor Jonny Bowden, Nutritionist, and author of The Great Cholesterol Myth, 2020.  

Dr. Zoe Harcombe, Ph.D. obesity researcher wrote her thesis on the lack of evidence behind the US Dietary Guidelines.  

Professor Andrew Mente, McMaster University, and researcher on the PURE Study. 

Alyssa Gallagher, Registered Dietician, Certified Diabetes Educator at Humphries Diabetes Center in Boise, Idaho. 

Doug Reynolds, Founder of LowCarb USA.

A warning about NAC for those who have emphysema or COPD

Lung adenocarcinoma development in NAC-treated mice.

Lung tissue histology in aged NAC-treated animals revealed the formation of tumors exhibiting lung adenocarcinoma characteristics in 50% of JunD–/– mice and in 10% of controls (Figure 4, A and B). The appearance of the tumors was typical of adenocarcinoma with conspicuous papillary structures associated with a collagen network that stained with Sirius Red, as reported in previous studies (Figure 4, C and D, and ref. 15). Adenocarcinoma tissue sections were also characterized by high counts of Ki67-positive cells compared with nonadenocarcinoma tissue (Figure 4E). In contrast, p21 and p16 staining activities were not detected in adenocarcinoma tissue (Figure 4F). No such tumors were detected in vehicle-treated JunD–/– or control mice, even when studied until 24 months of age. No animals in any group had tumors detected in the liver, spleen, or kidneys. Moreover, in situ studies detected no associated lesions in mice with lung cancer. Inflammatory infiltrates were detected in the lungs of most of the aged mice (Figure 4G) and were more marked in the aged JunD–/– mice than in their controls but were not affected by NAC treatment (Figure 4G). These changes were not related to alterations in lung levels of mucin (16), which did not differ between aged and young mice and which was not affected by JunD  inactivation (Supplemental Figure 2).

Definitely interesting and well-worth a conscious pause in the use of NAC.  Other antioxidants are available that seemingly have no increased risk of tumor growth.  

Cell senescence is known to inhibit cell transformation and tumor initiation. We consequently hypothesized that NAC treatment promoted tumor initiation by inducing escape from cell senescence (1718). Interestingly, p16- and p21-stained cells, although sparsely distributed in lungs from NAC-treated mice, were not seen within tumors (Figure 4F). In contrast, Ki67-stained cells were seen only within tumors (Figure 4B). JunD–/–-derived mouse embryonic fibroblasts (MEFs) display p53-dependent premature senescence (19), and NAC treatment decreases p53 levels in the context of lung tumor progression in mice (8). Accordingly, we found decreased lung p53 protein and p16 mRNA levels in NAC-treated aged mice (Supplemental Figures 3 and 4), supporting the concept that NAC limits the expression of key senescence effectors and tumor suppressor genes

 

Tuesday, July 5, 2022

Mexico President: Free Assange Or Dismantle Statue Of Liberty

REAL FOOD, IN CASE YOU NEEDED A REMINDER