And just out. As I have been saying. Spike = Neurodegeneration. This is very important. Please read. https://t.co/qCl0jZgPd4
— Walter M Chesnut (@Parsifaler) April 1, 2022
Okay, this is not good. None of the news coming from vaccinated individuals is good. It's going to require a lifetime of care. The spike protein crosses the blood brain barrier, BBB, and produces cognitive deficits and psychiatric problems. Meaning that you'll lose your mind unless you slow this process down by consuming neuroprotective nutrients, like fish oils, like the whole spectrum of B vitamins. B5 for youthfulness and longevity. B6 for healthy micro gut biome. B12 for nerve regeneration. Far-soluble B1 [Allithiamine, lipothiamine, and or benfotiamine] to protect your autonomic nervous system that automatically regulates all of your vital organs. See this post. What follows is extracted from the tweet above.
People worldwide are currently suffering from the coronavirus disease 2019 (COVID-19) pandemic caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2)1,2. Increasing evidence has shown that COVID-19 patients not only manifest respiratory-related symptoms, but also develop neurological and psychiatric symptoms, depending on the stage of infection, ranging from headache to cognitive and mood disorders3,4. According to clinical studies, 19% and 14% of COVID-19 patients develop depression and anxiety, respectively5 and 10–20% suffer from cognitive impairment6. Therefore, it is obvious that SARS-CoV-2 somehow affects the central nervous system (CNS), but the molecular and cellular mechanisms are still elusive. Previous studies suspected direct SARS-CoV-2 infection into the CNS, as SARS-CoV-2 spike protein and transcripts were detected in post-mortem brains. Then, as a port of CNS entry, SARS-CoV-2 invasion via olfactory receptor neurons was proposed7. However, a recent study using unbiased transcriptome analysis of the post-mortem brain tissue of COVID-19 patients did not succeed in detecting molecular traces of SARS-CoV-2 virus in the brain parenchyma8 negating direct SARS-CoV-2 infection into the CNS parenchyma. More recently, it was reported that intravenously administered radiolabeled S1 subunit of SARS-CoV-2 spike protein (S1 protein) can translocate into brain parenchyma by crossing the blood–brain barrier9. Therefore, this suggests the possibility that S1 proteins translocated into the brain parenchyma may affect brain functions, which might underlie the neurological or psychiatric symptoms of COVID-19 patients. The possibility was examined by introducing S1 proteins into mouse brains. We showed that the injection of S1 protein into mouse hippocampus induced cognitive deficits and anxiety-like behaviors. As mechanisms, we found that SARS-CoV-2 S1 protein exerted non-cell autonomous hippocampal neuronal cell death by inducing interleukin-1 beta (IL-1β) expression from glial cells
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