— healthbot (@thehealthb0t) March 1, 2022
GET NUTRITION FROM FARM-DIRECT, CHEMICAL-FREE, UNPROCESSED ANIMAL PROTEIN. SUPPLEMENT WITH VITAMINS. TAKE EXTRA WHEN NECESSARY
Tuesday, March 1, 2022
Want to wreak havoc on your metabolism? Then try ASPARTAME!!!
THINK!
— Walter M Chesnut (@Parsifaler) February 16, 2022
THE SPIKE PROTEIN AS AEROSOLIZED ASPARTAME: From food supply mechanisms to airborne pathogen
Let's not forget the nefarious history of this substance.
Not everyone drank it. Too slow?
The BBB issues led me here.
Phenylalanine and COVID-19. https://t.co/4QsllOpK9h pic.twitter.com/2mvU5ING4C
Monday, February 28, 2022
"Studies suggest beneficial effects of azithromycin in reducing viral load of hospitalized patients, possibly interfering with ligand/CD147 receptor interactions"
Dr. Ryan Cole explains how the spike protein weakens the immune system and could cause susceptibility to other viruses and exacerbate cancer.
— The Vigilant Fox 🦊 (@VigilantFox) November 23, 2021
"I'm seeing these very calm, manageable cancers take off like wildfire, like hydras, like dragons, and they're going everywhere. pic.twitter.com/XctUtiodRN
From NCBI,
CD147, a receptor on host cells, is a novel route for SARS-CoV-2 invasion. Thus, drugs that interfere in the spike protein/CD147 interaction or CD147 expression may inhibit viral invasion and dissemination among other cells, including in progenitor/stem cells. Studies suggest beneficial effects of azithromycin in reducing viral load of hospitalized patients, possibly interfering with ligand/CD147 receptor interactions; however, its possible effects on SARS-CoV-2 invasion has not yet been evaluated. In addition to the possible effect in invasion, azithromycin decreases the expression of some metalloproteinases (downstream to CD147), induces anti-viral responses in primary human bronchial epithelial infected with rhinovirus, decreasing viral replication and release. Moreover, resident lung progenitor/stem are extensively differentiated into myofibroblasts during pulmonary fibrosis, a complication observed in COVID-19 patients. This process, and the possible direct viral invasion of progenitor/stem cells via CD147 or ACE2, could result in the decline of these cellular stocks and failing lung repair. Clinical tests with allogeneic MSCs from healthy individuals are underway to enhance endogenous lung repair and suppress inflammation.
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