CONCERNED CITIZEN: This is a Liver Fluke - found in a Human Stomach.

This is a Liver Fluke - found in a Human Stomach.

And yes they were before you ask….. pic.twitter.com/qft5DzIiOV

— Concerned Citizen (@BGatesIsaPyscho) November 25, 2024

Ah, wonderful, a liver fluke.  What, pray tell, is a liver fluke?  A parasite, a worm found in the liver.  

Healthline explains,

A liver fluke is a parasitic worm. You can become infected after eating contaminated raw or undercooked freshwater fish or watercress. Infections are usually treated with a drug called triclabendazole.

After liver flukes have been ingested, they travel from your intestines to your bile ducts in your liver where they then live and grow.

Although most infected individuals don’t show any symptoms, sometimes symptoms arise related to the biliary system. In rare cases, long-term complications can also develop.

Liver fluke infections aren’t common in the United States, but they do occur. Your risk of infection increases if you travel to parts of the world where the parasites are widespread.  

Triclabendazole sound like an antiparasitic like Fenbendazole.  Other parasitics exist.  Garlic, Cayenne Pepper, nicotine. 

FenLAB 222 | mg | >99% | 90 | ct https://t.co/oJ59TCHMQu via @amazon

— St. Michael, the Archangel (@aveng_angel) November 26, 2024

Neck, Liver, Bladder, and Pancreas Cancers in Joe Tippens cured 3 months after taking Fenbendazole

Joe Tippens a cancer patient who was given three months to live, took a combination of nutrients including Fenbendazole, while deciding not to change his diet & it worked.

• Fenbendazole (222mg)
• Vitamin E (800IU daily)
• Curcumin (600mg daily)
• CBD oil (25mg per day)… pic.twitter.com/hWDBzrQhiD

— Barbara Oneill (@BarbaraOneillAU) May 21, 2024

How Fenbendazole works?

How Fenbendazole works? - Fenbendazole resource https://t.co/J3FsAmfygR

— Kelly DNP🐭Functional/Integrative Med (@kacdnp91) January 14, 2024 

 

From Fenbendazole.org.

. . . there are 3 main mechanisms by which fenbendazole kills cancer:

1) Apoptosis induction. The anti-tumor effect is believed to be through the interaction of the drug with the β-tubulin leading to cell cycle arrest and cytotoxic.

Inhibition of polymerization of tubulin into microtubules by benzimidazole carbamates in both helminths and human tumor cells is well documented. (Source1, Source2)

2) Inhibition of glucose uptake in cancer cells. Malignant cells are known to have an enormous glucose uptake. Cancer cells normally consume glucose 200 times faster than ordinary cells due to aerobic glycolysis (Warburg effect). This can be seen in PET scans – the metabolically active sites, which use more radioactive glucose can be clearly seen and are generally thought to be cancerous tumors or sites of inflammation.

Fenbendazole limits cancer cell fueling with sugar by limiting glucose uptake, decreasing the amount of GLUT transporters (canals that take glucose into the cancer cells from the blood), and the enzyme hexokinase 2. The latter is important for cancer cell survival, it helps the tumors thrive by generating more sugar and accelerating lactic acidosis in the extracellular matrix. (Source1, Source2) 

3) Reactivation of the p53 gene. This work mechanism is still relatively controversial, and more studies need to be done to confirm that Fenben causes this action. (Source)

However, there is an increasing number of studies that confirm the fact that fenbendazole might truly increase the strongest tumor suppressor in our bodies – p53. (Source)

It is known that elephants have 20 copies of the p53 gene in their genome and humans have only 1 copy. Interestingly, that is why elephants get cancer less frequently than us, humans. Despite these creatures having a huge body size, a larger number of cells, and a bigger number of potential genetic mutations.