Babies receive 250 mcg of aluminum just after birth, 1,225 mcg of aluminum at 2 months of age, and 4,925 mcg of aluminum by 18 months of age via the adjuvants in their childhood shots.
— Danny Zeck (@d_a_zeck) March 17, 2022
Where is the proof that aluminum hydroxide and aluminum phosphate are safe *when injected?*
As a neurotoxin, aluminum dulls the mind. How in God's green earth could parents sacrifice their babies to such a degraded life by handing them over to a schedule of vaccines? Exposing your babies to mumps, measles, and others will only strengthen his immune system. Vaccines, with aluminum as an adjuvant, cripple the immune system, the mind, and nervous system in general. NCBI explains that
Aluminum is regularly taken up with the daily diet. It is also used in antiperspirants, as an adjuvant for vaccination, and in desensitization procedures.
Aluminum reduces neuro-plasticity.
Aluminum (Al3+) exhibits a high affinity to proteins, which it is able to cross-link. In contrast to other ubiquitously occurring metals such as iron, manganese, and zinc, aluminum is not known to perform a physiological function in the human organism (23). Clinically relevant, neurotoxic effects have been described in dialysis patients. Aluminum salts, which were formerly added to the dialysate as a phosphate binder, were identified as the causal agents (15). Patients exhibited elevated aluminum concentrations in plasma and brain tissue (15, 24). Those affected showed disorientation, memory impairments, and, at advanced stages, dementia (15). The cause of these effects lies, firstly, in the slow—compared with other organs—removal of aluminum from the brain (e11) and, secondly, in the multitude of biological processes affected by aluminum in the brain (23).
In addition to inducing oxidative stress and binding to negatively charged membrane structures in neurons, aluminum is able to modify hippocampal calcium signal pathways that are crucial to neuronal plasticity and, hence, to memory (e12). Cholinergic neurons are particularly susceptible to aluminum neurotoxicity, which affect synthesis of the neurotransmitter acetylcholine (e13). Particularly the latter two neurobiological effects are also relevant in the presumed association between aluminum and Alzheimer’s disease (the Alzheimer’s hypothesis) (23). Aluminum-related neurotoxic effects could be partially reversed once aluminum contamination was no longer present in the dialysate (15).
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